Misconceptions that testosterone causes prostate cancer are not new, but the startling truth is that it doesn’t, in fact, higher levels of testosterone decreases the size of prostate. There is no one culprit to what causes prostate cancer other than changes in the DNA of a normal prostate cell that amount to inherited or acquired gene mutations.
Despite decades of research, there is no compelling evidence that testosterone has a causative role in prostate cancer. There is no compelling evidence at present to suggest that high testosterone levels or testosterone administration increases the risk of cancer. Prostate cancer becomes more relevant at the time of a man’s life when testosterone levels decline. Experienced clinicians aim for the upper-normal range, in order to optimize treatment.” New England Journal of Medicine 2004; 350: 482-92
The prevalence of prostate cancer in men with low testosterone levels is substantial in comparison with high testosterone levels. Lower testosterone levels increase risk of prostate cancer and cancer severity.” New England Journal of Medicine 2004; 350: 482-92
If anything, testosterone replacement triggers the following advantages:
- Improved Erectile Function: Testosterone directly affects reproductive functions. Sexual functions may be decreased when testosterone is deficient and improved when testosterone levels are brought back to normal.
- Reduced Hair Thinning: Testosterone assists hair follicles in the producing of hair, and cells can die away due to a lack of hormonal production and dryness.
- Restored Muscle Mass: When testosterone levels decline, men can lose between 12 or 15 pounds of lean muscle mass. Restoring testosterone to healthy levels improves the ability of muscles to regenerate with exercise.
To reiterate, prostate cancer is not and isn’t associated to higher testosterone levels from testosterone replacement. The process of testosterone replacement even backtracks the risk of prostate cancer to a less risk or even to a normal and benign level of testosterone.
